25.11.2022; Vortrag
K. Schneider - A profound attenuation of auditory transients in the magnocellular medial geniculate nucleus in dyslexia is a potential neuroimaging diagnostic in individual subjects
Abstract
Hypotheses concerning the neurological basis of dyslexia include dysfunction of the magnocellular system in the brain, abnormal temporal processing, and/or deficient phonological skills. Each of these theories has merit, but the link among them has not been identified. Here we show that people with dyslexia have a selective impairment in the magnocellular portion of the medial geniculate nucleus, the auditory relay in the thalamus. We used functional magnetic resonance imaging to measure highly consistent and profoundly attenuated responses to non-linguistic transient but not sustained sounds compared to normal readers. This measurement is a 95% accurate diagnostic for dyslexia in individual subjects. Our finding identifies a core deficit causing dyslexia that can unify the three hypotheses.
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